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Other researchers have proposed that these cellular changes are the result of “chronic cerebral hypoperfusion” facilitated by hypertension, atherosclerosis, diabetes, hypercholesterolemia and aging [11-14]

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Additional mechanistic studies demonstrated that both dihexa and Nle 1 -AngIV, its parent compound, were effective stimulators of hippocampal synaptogenesis, thus providing a rational explanation for their procognitive activities