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The Mthfr −/− mice were with none MTHFR enzyme activity in all tissues, whereas the Mthfr +/− showed 60% residual activity, similar to the value observed in patients homozygous for the C667T polymorphism 40

BOGOF

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This review summarizes the various cellular events involved in AIH and discusses their potential therapeutic targets, with the aim of providing new ideas for the treatment of AIH