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Together, these studies show that inhibition of lipogenic pathways protects from MASLD in multiple models with decreased levels of lipogenic genes, and that GSH depletion through Chac1 overexpression recapitulates similar protective effects by broadly downregulating lipogenic genes

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In muscle tissue, transamination of BCAAs yields glutamic acid, which in turn contributes anaplerotic substrates (such α-ketoglutaric acid) to the Krebs cycle, while simultaneously generating NADH to fuel mitochondrial respiration 16