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For instance, DCA can activate plasmacytoid DCs to induce type I IFNs via TLR7/MyD88 signaling ( Other BAs like TUDCA modulate the DC microenvironment indirectly, while 3-oxoLCA and isoLCA inhibit Th17 cells, synergizing with isoDCA-driven Treg promotion ( Thus, the gut BA pool regulates DC function via FXR, TGR5, and Toll-like receptors (TLRs), driving either tolerance or activation depending on context

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